Drug resistance patterns of herpes simplex virus isolates from patients treated with acyclovir.

Drug resistance patterns of herpes simplex virus isolates from patients treated with acyclovir.

Drug resistance patterns of herpes simplex virus isolates from patients treated with acyclovir.
These days, it isn’t uncommon to hear about certain “miracle foods” that come in handy for just about anything. The ACV-resistant isolate from patient 1 was cross-resistant to dihydroxypropoxymethylguanine and bromovinyldeoxyuridine, but still susceptible to three fluoro-substituted pyrimidines, 2′-fluoro-5-iodo-1-beta-D-arabinofuranosylcytosine (FIAC), 2′-fluoro-5-iodo-1-beta-D-arabinofuranosyluracil (FIAU), and 2′-fluoro-5-iodo-1-beta-D-arabinofuranosylthymine (FMAU). Together, apple cider vinegar and honey are a powerful health booster. Recombination experiments among all combinations of the nine MCMV mutants were performed and supported the complementation data in that no recombination could be detected. An isolate obtained during a subsequent recurrence in patient 2 was susceptible to ACV and the other TK-dependent agents. Herpes simplex virus-1 (HSV-1) and Herpes simplex virus-2 (HSV-2) are DNA viruses belonging to the Herpesviridae family that cause widespread infection in humans. In none of the isolates studied was a change in sensitivity to phosphonoformic acid observed.

Just add two teaspoons of apple cider vinegar and two teaspoons of honey to a glass of water and drink up. These findings indicate that mixed patterns of drug-resistance to TK-dependent antiviral compounds can occur in clinical isolates, resulting from changes in either the amount or the affinity of viral TK activity. I no longer had bad breath in the morning (kind of an unexpected result) If you’d like to try what I did out for yourself, here’s how I did it. These results suggest that susceptibility of MCMV to ACV is confined to a product of a single gene and that a mutation of this gene can lead to an altered phenotype when compared with parent virus in susceptibility of DNA synthesis to PAA, ara-A, and aphidicolin, drugs that are known to inhibit DNA polymerase activity. Links to PubMed are also available for Selected References.

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