Neurological diseases caused by neuropathogenic strains of EHV1 are being reported with increasingfrequency. and L. Equine urine has a mucoid, tenacious content unique to the species that may help to lubricate and protect the luminal surfaces of the tract from scabrous damage from crystalluria. As one calendar year draws to a close and another begins, many people resolve to take steps to improve their lives. The CNS of all cases was negative by RT-PCR or immunohistochemistry (IHC) for African horse sickness (AHS), equine encephalosis virus, equine herpes viruses 1 and 4, other zoonotic flaviviruses, alphaviruses, and shunivirus, and either by immunofluorescence or IHC for rabies. After the horse, who was trained by Jacqueline Falk, died, the rest of the horses in the same barn were placed in isolation and they were not allowed to train during regular hours or be entered to race. One of those diseases is equine viral arteritis (EVA).
18 to table a bill authorizing one casino to be located at Rockingham Park, dealing yet another blow to hopes for the revitalization of the track and the return of live Thoroughbred racing. She’s reject… The Massachusetts Gaming Commission said no dice to the state’s Thoroughbred racing and breeding industry when it cast a preliminary 3-1 vote Sept. Bladders that become paralyzed because of these pathologic processes often show deposition of a sandy, crystalloid sediment, a condition called sabulous cystitis, instead of formed uroliths. Bladder paralysis permits settling of deposits onto the bladder floor, where the material’s weight prevents it from being extruded during dribbling of urine from the incontinent bladder. Nothing changed for the virus from the pain disappears even if there is sometimes subscribe today! Analysis of the mineral content of sabulous sediment in horses has shown the main component to consist of calcium carbonate composed of calcite and vaterite, and magnesium, sodium, potassium, reduced iron, copper, manganese, and traces of phosphates, sulfates, and silicates.
Careful neurologic examination may help differentiate whether there are upper motor neuron or lower motor neuron signs affecting the bladder, although horses with this problem are typically presented for assessment well into the chronic stage of dysfunction,when clinical findings tend to blur. This sample along with a blood sample should be sent in together for analysis. Palpation of the bladder per rectum may reveal moderate distension, because the proximal urethral sphincter will be tonic. the only thing i don’t know is how airborne it could go. Once the disease becomes chronic, however, clinical distinction between the original type of neurologic deficit becomes difficult to impossible, as the ongoing presence of sabulous sediment and attendant inflammation lead to progressive loss of detrusor function and eventual signs of complete bladder atony. Although some horses with paralytic bladders will show additional signs of neurologic injury such as ataxia, flaccid tail or anal tone, or denervation atrophy of hindquarter muscle groups, other horses may be presented for evaluation in which no additional neurologic deficits can be determined. Gloria, I have no expertise to offer you, but when I read your post, my heart ached.
The condition is diagnosed via cystoscopic visualization of the bladder interior following evacuation of urine from the lumen. Cystic calculi can be palpated with transrectal ultrasound, but cystoscopy is preferred. I can’t bring him home, of course. The remainder of the urinary tract should be further examined. This rapidly progressing disease is the single most important cause of fatal sporadic encephalitis in the US. The tail head was elevated and deviated slightly to the left. Placement of an indwelling catheter for a brief initial period helps lower urine stasis and maintains the bladder in a fairly decompressed state, abating further detrusor damage.
Adding acetic acid to decrease luminal pH during lavage is advocated. I usually recommend 8 treatments at 1-3 day intervals. Salt should be provided, to increase water consumption and fluid diuresis. In all cases, provide owners with a guarded to negative prognosis for recovery of normal function in horses in which incontinence has been present for several months or longer. comm.). Fillies were housed in a communal paddock and colts in an adjacent paddock, and treatment and placebo groups were commingled. Bethanechol (20-40 mg or 0.07 mg/kg SQ q 8 hours, 80 mg PO q 8 hours do not give I.V.
or I.M.) is not currently available as a proprietary formulation but can be obtained from compounding pharmacies. Phenoxybenzamine, (0.2-0.7 mg/kg PO, q 6-8 hrs) is an adrenergic antagonist, and may be given to help relax the distal urethral sphincter in cases where the condition is determinedly due to upper motor neuron damage. Phenazopyridine (4 mg/kg PO q 8-12 hours), an azo dye compound that acts to confer relief from irritation or spasm of the urinary tract mucosa via local anesthetic activity, may also be administered in the initial management of the cystitis caused by the sedimentary accumulation. In accordance with FDA guidelines, we not recommend Transfer Factor for any specific condition. Owners should be warned that it will stain skin and textiles which inadvertently come into contact with the substance. In humans with urinary tract inflammation the agent alleviates symptoms of dysuria, frequency, burning, and the sensation of urgency. Mares in which urinary incontinence is from hypoestrogenism may enjoy a better prognosis for response to treatment.
Successful management of the condition in such mares has been observed in association with administration of estradiol cypionate or benzoate (5-10 _g/kg I.M. A horse can be “colicky” for many reasons—large colon torsions, small intestinal strangulations, spasmodic ep… Approximately 20,000 mares are bred in Kentucky each year, which means that it is literally “raining foals” in the spring, as long as things go as planned. Saunders Co., pp. 597-601. * Diaz-Espineira M, Escolar E, Bellanato J et al: Infrared and atomic spectrometry analysis of the mineral composition of a series of equine sabulous material samples and urinary calculi. Res Vet Sci Jul-Aug; 63(1):93-5, 1997.
* Mandell GL and Petri WA: Antimicrobial agents, in Hardman JG, Gilman AG, Limbird LE et al, eds.: Goodman & Gilman’s The pharmacologic basis of therapeutics, 9th ed., New York, 1996, McGraw-Hill, p. 1070.